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First-Rate,
First-Line Immune Defense
When your throat gets scratchy, you feel feverish,
or you see a red welt developing around that tiny splinter, you are seeing
the effects of a highly sophisticated immune defense strategy. Symptoms
of a sickness tell us that our immune defenses have mounted a counterattack
against whatever infectious agent has entered our body. In order to work
well, our immune system must operate like a well-oiled machine.
Much like the workings of sensitive radar, the
immune system must recognize germs as foreign intruders and separate them
from the body’s own cells and molecules. Because it works on a molecular
level, immune cells work as part of a highly complex and reactive system.
The
immune system’s ability to respond properly determines our very health
and survival.
When bacteria, viruses, fungi, toxins or cancer
cells invade, the immune system springs into action, seeking out and eradicating
the infectious enemy. Immune system agents are activated to neutralize
potentially dangerous microbes. In addition, other immune cells act as
messengers, releasing molecules that work to fine tune the whole process.
Whenever our immune system successfully foils
an infection, it emerges stronger and better equipped to fight future health
threats.
The Antigen Factor
All cells, including infectious invaders, carry
identifiable antigens on their surface. An antigen is any substance that
causes the immune system to produce antibodies. Much like individual fingerprints,
these specific molecular signatures tell the immune system whether the
cell is friend or foe. This is why transplanted organs are often attacked
by the recipient’s immune system. Their foreign antigen codes are translated
as intruder cells so the immune system mounts its attack. It is estimated
that our bodies can react to over 100 million different antigens.
While our immune system routinely sifts and sorts
through trillions of cells and their antigen codes, it may be lacking as
many as 50,000 specific antigen responses. Without immune awareness of
those missing cellular imprints, we become more vulnerable to those specific
invaders. In addition, keep in mind that many infectious agents routinely
mutate, fooling the immune systems. This is why we can catch multiple colds
and flus over the course of time.
We already possess our own innate storehouse of
immune molecules called transfer factors that occupy our T lymphocytes.
These factors enable the T cells of our immune system to set off immediate
alarms when certain antigens are identified as undesirable. So, you can
see why adding to our already existing stockpile of transfer factors would
be a good thing.
The
Key: A Maximized Immune Messaging System
Transferring the right information from one group
of immune cells to another helps to prevent each of the four threats mentioned
previously.
The right molecular messengers can instruct, coordinate,
activate and suppress immune cells according to need.
We usually operate with information collected
from our own immune experience or exposure, but we can do much better than
that. Our immune system needs maximum access to molecular information whether
it comes from our own internal data banks or whether it is imported from
other sources. The point is that whether generated from within or from
without, immune messenger molecules in the form of transfer factors all
speak the same language.
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